Relapse in children with acute lymphoblastic leukaemia involving selection of a pre-existing drug resistant sub-clone Short title: Clonal selection and relapse in ALL

نویسندگان

  • Seoyeon Choi
  • Michelle J. Henderson
  • Edward Kwan
  • Alex H. Beesley
  • Rosemary Sutton
  • Anita Y. Bahar
  • Jodie Giles
  • Nicola C. Venn
  • Luciano Dalla Pozza
  • David L. Baker
  • Glenn M. Marshall
  • Ursula R. Kees
  • Michelle Haber
  • Murray D. Norris
چکیده

Children’s Cancer Institute Australia for Medical Research, Sydney, Australia* Telethon Institute for Child Health Research, and Centre for Child Health Research, University of Western Australia, Perth, Australia Oncology Unit, The Children's Hospital, Westmead, Sydney, Australia Department of Haematology–Oncology, Princess Margaret Hospital, Perth, Australia Centre for Children’s Cancer and Blood Disorders, Sydney Children’s Hospital, High St, Randwick, Sydney, Australia

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Relapse in children with acute lymphoblastic leukemia involving selection of a preexisting drug-resistant subclone.

Relapse following remission induction chemotherapy remains a barrier to survival in approximately 20% of children suffering from acute lymphoblastic leukemia (ALL). To investigate the mechanism of relapse, 27 matched diagnosis and relapse ALL samples were analyzed for clonal populations using polymerase chain reaction (PCR)-based detection of multiple antigen receptor gene rearrangements. These...

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Relapse in pediatric T-cell acute lymphoblastic leukemia (T-ALL) remains a significant clinical problem and is thought to be associated with clonal selection during treatment. In this study we used an established pre-clinical model of induction therapy to increase our understanding of the effect of engraftment and chemotherapy on clonal selection and acquisition of drug resistance in vivo. Immu...

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In acute myeloid leukaemia (AML) initiating pre-leukaemic lesions can be identified through three major hallmarks: their early occurrence in the clone, their persistence at relapse and their ability to initiate multilineage haematopoietic repopulation and leukaemia in vivo. Here we analyse the clonal composition of a series of AML through these characteristics. We find that not only DNMT3A muta...

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تاریخ انتشار 2007